Vasodilation and Blood Flow Dynamics
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In brief: FIR exposure promotes vasodilation and increased blood flow through endothelial signaling and nitric oxide-mediated pathways.
When tissues absorb FIR radiation, endothelial cells lining blood vessels respond by upregulating nitric oxide synthase (eNOS) activity.
[15]
This results in increased production of nitric oxide (NO), a potent vasodilator that relaxes smooth muscle cells within the vascular wall.
[15]
FIR-induced vasodilation improves blood flow through several coordinated mechanisms:
- Relaxation of vascular smooth muscle cells
- Reduction in peripheral vascular resistance
- Increased capillary perfusion within treated tissues
- Improved blood rheology and flow velocity
Multiple clinical investigations using laser Doppler flowmetry have quantified this effect, documenting increases in cutaneous blood flow by 30–60% during FIR exposure, even in subjects at complete rest (Yu et al.).
[21]
Importantly, this vasodilatory response occurs without a corresponding increase in heart rate or systemic blood pressure, indicating a localized microvascular effect rather than generalized cardiovascular stress.
[21]